Search results for " atrophic"

showing 10 items of 19 documents

Ikivėžinių skrandžio gleivinės pokyčių paplitimas Helicobacter pylori sukelto lėtinio gastrito metu tarp Rytų Europos (Lietuvos, Latvijos) ir Azijos …

2007

The aim of the study was to compare the prevalence and severity of precancerous condition – gastric atrophy and intestinal metaplasia (IM) between Eastern European (Lithuania and Latvia) and Asian (Taiwan) countries in population older than 55 years. Methods. Patients aged 55 years and older, referred for upper endoscopy due to dyspeptic symptoms, were included in the study. Gastric biopsies were histological investigated according modified Sydney classification. Helicobacter pylori (H. pylori) was detected if any two of three methods (urease test, histology, and serology) were positive. Results. Overall 322 patients included: 52 from Taiwan (TW), 171 from Latvia (LV) and 99 from Lithuania …

Gastritis AtrophicMalemedicine.medical_specialty616.3 [udc]Helicobacter pylori ; Gastritis atrophic ; Intestinal diseases ; Metaplasia ; Precancerous conditionsBiopsyPopulationTaiwanRapid urease testintestinal metaplasiaGastroenterologyHelicobacter InfectionsAtrophyStomach NeoplasmsMetaplasiaInternal medicineGastroscopyPrevalenceHumansMedicineeducation616.33/.34 [udc]AgedMetaplasiaeducation.field_of_studyHelicobacter pyloribiologybusiness.industryStomachgastritisIntestinal metaplasia<em>Helicobacter pylori</em>; gastritis; atrophy; intestinal metaplasiaLithuaniaGeneral MedicineMiddle AgedHelicobacter pylorimedicine.diseasebiology.organism_classificationLatviaEastern europeanFemaleAtrophymedicine.symptomGastritisbusinessPrecancerous Conditions<em>Helicobacter pylori</em>
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Uncommon oral manifestation of lichen sclerosus: critical analysis of cases reported from 1957 to 2016

2017

Background Lichen sclerosus is a mucocutaneous autoimmune disease which might be initiated by infectious pathogens as Borrelia Bugrdorferi and HPV. This disease shows destructive potential and is rarely diagnosed in oral mucosa. The purpose of this paper is to evaluate the characteristics of cases described in literature from 1957 to 2016, looking to provide valuable evidence about clinicopathologic features of this disease. Material and Methods A MedLine search was performed aiming to find oral lichen sclerosus cases in literature and discuss its demographical and pathological characteristics as well as treatment methods performed for these cases. Results 34 oral lichen sclerosus cases wit…

AdultMalemedicine.medical_specialtyTime FactorsAdolescentMucocutaneous zoneReviewDiseaseLichen sclerosusAsymptomaticYoung Adult030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicinestomatognathic systemmedicineHumansOral mucosaYoung adultChildGeneral DentistryPathologicalAgedAutoimmune diseaseOral Medicine and Pathologyintegumentary systembusiness.industryMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseDermatologystomatognathic diseasesLichen Sclerosus et Atrophicusmedicine.anatomical_structureOtorhinolaryngology030220 oncology & carcinogenesisUNESCO::CIENCIAS MÉDICASFemaleSurgerymedicine.symptomMouth DiseasesbusinessMedicina Oral Patología Oral y Cirugia Bucal
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Association of Long Non-Coding RNA Polymorphisms with Gastric Cancer and Atrophic Gastritis

2020

Long non-coding RNAs (lncRNA) play an important role in the carcinogenesis of various tumours, including gastric cancer. This study aimed to assess the associations of lncRNA ANRIL, H19, MALAT1, MEG3, HOTAIR single-nucleotide polymorphisms (SNPs) with gastric cancer and atrophic gastritis. SNPs were analyzed in 613 gastric cancer patients, 118 patients with atrophic gastritis and 476 controls from three tertiary centers in Germany, Lithuania and Latvia. Genomic DNA was extracted from peripheral blood leukocytes. SNPs were genotyped by the real-time polymerase chain reaction. Results showed that carriers of MALAT1 rs3200401 CT genotype had the significantly higher odds of atrophic gastritis …

0301 basic medicineGastritis AtrophicMalemedicine.medical_specialtylcsh:QH426-470GenotypeAtrophic gastritisSingle-nucleotide polymorphismmedicine.disease_causeGastroenterologyPolymorphism Single NucleotideArticleTertiary Care Centers03 medical and health sciences0302 clinical medicineGene FrequencyStomach NeoplasmsInternal medicineGermanyatrophic gastritisGenotypeGeneticsmedicineOdds RatioHumansGenetic Predisposition to DiseaseRNA NeoplasmGenetics (clinical)AllelesAgedMALAT1long non-coding RNAbusiness.industrylong non-coding RNA ; single-nucleotide polymorphism ; gastric cancer ; atrophic gastritisgastric cancerCancerHOTAIRMiddle Agedsingle-nucleotide polymorphismmedicine.diseaseLong non-coding RNAlcsh:Genetics030104 developmental biology030220 oncology & carcinogenesisFemaleRNA Long NoncodingCarcinogenesisbusinessGenes
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Analysis of thiamine transporter genes in sporadic beriberi

2014

Abstract Objective Thiamine or vitamin B 1 deficiency diminishes thiamine-dependent enzymatic activity, alters mitochondrial function, impairs oxidative metabolism, and causes selective neuronal death. We analyzed for the first time, the role of all known mutations within three specific thiamine carrier genes, SLC19 A2, SLC19 A3 , and SLC25 A19 , in a patient with atrophic beriberi, a multiorgan nutritional disease caused by thiamine deficiency. Methods A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema, a subacute sensorimotor neuropathy, and incontinence. Despite normal vitamin B 1 serum levels, his clinical picture was rapidly reverted by high-dose in…

AdultMalemedicine.medical_specialtySLC19 A- SLC25 A19SLC19 AEndocrinology Diabetes and MetabolismGene mutationBeriberimedicine.disease_causeMitochondrial Membrane Transport Proteinslaw.inventionBeriberilawInternal medicineGenotypemedicineThiamine transporterObjective: Thiamine or vitamin B1 deficiency diminishes thiamine-dependent enzymatic activity alters mitochondrial function impairs oxidative metabolism and causes selective neuronal death. We analyzed for the first time the role of all known mutations within three specific thiamine carrier genes SLC19 A2 SLC19 A3 and SLC25 A19 in a patient with atrophic beriberi a multiorgan nutritional disease caused by thiamine deficiency. Methods: A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema a subacute sensorimotor neuropathy and incontinence. Despite normal vitamin B1 serum levels his clinical picture was rapidly reverted by high-dose intramuscular thiamine treatment suggesting a possible genetic resistance. We used polymerase chain reaction followed by amplicon sequencing to study all the known thiamine-related gene mutations identified within the Human Gene Mutation Database. Results: Thirty-seven mutations were tested: 29 in SLC19 A2 6 in SLC19 A3 and 2 in SLC25 A19. Mutational analyses showed a wild-type genotype for all sequences investigated. Conclusion: This is the first genetic study in beriberi disease. We did not detect any known mutation in any of the three genes in a sporadic dry beriberi patient. We cannot exclude a role for other known or unknown mutations in the same genes or in other thiamine-associated genes in the occurrence of this nutritional neuropathy.HumansThiamineGenePolymerase chain reactionGeneticsMutationNutrition and DieteticsbiologyMembrane Transport ProteinsThiamine Deficiencymedicine.diseaseAlcoholismEndocrinologyMutationbiology.proteinThiamineMutations
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Anti-parietal cell antibodies – diagnostic significance

2015

Anti-parietal cell antibodies (APCA) are an advantageous tool for screening for autoimmune atrophic gastritis (AAG) and pernicious anemia (PA). The target for APCA is the H+/K+ ATP-ase. It has been demonstrated, that APCA target both, the alpha, and beta subunits of the proton pump, although the major antigen is the alpha subunit. Circulating serum APCA can be detected by means of immunofluorescence, enzyme-linked immunosorbent assay – currently the most commonly used method, and radioimmunoprecipitation assay (RIA) – the 4A subunit has been optimized as a molecularspecific antigen probe. RIA is the most accurate method of antibody assessment, characterized by highest sensitivity. APCA can …

Atrophic gastritisAutoimmunityVitiligomedicine.disease_causeImmunofluorescenceAutoimmune DiseasesHelicobacter InfectionsAutoimmunity03 medical and health sciences0302 clinical medicineParietal Cells GastricAntigenmedicineHumansAntigensAutoantibodiespernicious anemiaParietal cellmedicine.diagnostic_testbiologybusiness.industryGeneral Medicinemedicine.diseasemedicine.anatomical_structure030220 oncology & carcinogenesisImmunologyPernicious anemiabiology.protein030211 gastroenterology & hepatologyAutoimmune atrophic gastritisAntibodybusinessAnti-parietal cell antibodiesAdvances in Medical Sciences
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The phenotype of gastric mucosa coexisting with Barrett's oesophagus.

2001

Barrett's oesophagus complicates the gastro-oesophageal acid reflux. Helicobacter pylori infection, particularly with cagA positive strains, induces inflammatory/atrophic lesions of the gastric mucosa, which may impair acid output. No systematic study has investigated the phenotype of the gastric mucosa coexisting with Barrett's oesophagus. This study was designed to identify the phenotype of gastric mucosa associated with Barrett's oesophagus.In this retrospective case control study, the phenotype of the gastric mucosa was histologically characterised in 53 consecutive patients with Barrett's oesophagus and in 53 (sex and age matched) non-ulcer dyspeptic controls. Both patients and control…

AdultGastritis AtrophicMalemedicine.medical_specialtyPathologyAtrophic gastritisBiopsySpirillaceaeBarrett's oesophagus gastritis in Barrett's oesophagus Barrett's oesophagus and gastric precancerous lesionsdigestive systemGastroenterologyHelicobacter InfectionsPathology and Forensic MedicineBarrett's oesophagus and gastric precancerous lesionsBarrett EsophagusInternal medicineotorhinolaryngologic diseasesGastric mucosamedicineHumansCagAAgedRetrospective StudiesAged 80 and overHelicobacter pyloribiologybusiness.industrygastritis in Barrett's oesophagusStomachdigestive oral and skin physiologyIntestinal metaplasiaGeneral MedicineMiddle AgedHelicobacter pyloribiology.organism_classificationmedicine.diseasedigestive system diseasesBarrett's oesophagussurgical procedures operativePhenotypemedicine.anatomical_structureGastric MucosaCase-Control StudiesPapersFemaleGastritismedicine.symptombusiness
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Lack of association between gene polymorphisms of Angiotensin converting enzyme, Nod-like receptor 1, Toll-like receptor 4, FAS/FASL and the presence…

2011

Abstract Background Several polymorphisms of genes involved in the immunological recognition of Helicobacter pylori and regulating apoptosis and proliferation have been linked to gastric carcinogenesis, however reported data are partially conflicting. The aim of our study was to evaluate potential associations between the presence of gastric cancer (GC) and high risk atrophic gastritis (HRAG) and polymorphisms of genes encoding Angiotensin converting enzyme (ACE), Nod-like receptor 1 (NOD1), Toll-like receptor 4 (TLR4) and FAS/FASL. Methods Gene polymorphisms were analyzed in 574 subjects (GC: n = 114; HRAG: n = 222, controls: n = 238) of Caucasian origin. ACE I/D (rs4646994), NOD1 796G&gt;…

AdultGastritis AtrophicMaleFas Ligand ProteinGenotypeAtrophic gastritisPeptidyl-Dipeptidase AWhite PeopleFas ligandHelicobacter InfectionsRisk FactorsStomach NeoplasmsNod1 Signaling Adaptor ProteinNOD1GenotypemedicineGeneticsHumansGenetics(clinical)fas ReceptorAllelesGenetics (clinical)AgedAged 80 and overPolymorphism GeneticHelicobacter pyloribiologyCancerAngiotensin-converting enzymeMiddle AgedHelicobacter pylorimedicine.diseasebiology.organism_classificationToll-Like Receptor 4ApoptosisImmunologybiology.proteinFemalePrecancerous ConditionsResearch ArticleBMC Medical Genetics
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Gastric plasma biomarkers and Operative Link for Gastritis Assessment gastritis stage

2011

Introduction The Operative Link for Gastritis Assessment (OLGA) staging system has been proposed as a histopathological reporting system of gastric atrophy. Noninvasive methods for indirect evaluation of gastric mucosal atrophy by biomarkers are also being introduced. Objectives To analyze gastric mucosal atrophy by biomarkers, pepsinogen I (PgI), pepsinogen II (PgII), PgI/PgII ratio, fasting gastrin-17 (G-17), stimulated gastrin-17 (sG-17), in relation to OLGA gastritis stage. Patients and methods Gastric biopsies were taken from 269 prospective patients referred for upper endoscopy because of dyspeptic problems and evaluated by two expert pathologists (D.J. and P.S.). Atrophy was assessed…

AdultGastritis AtrophicMalemedicine.medical_specialtyStage iiPlasma biomarkersGastroenterologyHelicobacter InfectionsAtrophyPepsinogen AInternal medicineGastrinsPepsinogen CHumansMedicineProspective StudiesStage (cooking)Prospective cohort studyAgedAged 80 and overHelicobacter pyloriHepatologybusiness.industryGeneral surgeryGastroenterologyMiddle Agedmedicine.diseaseFemaleGastritismedicine.symptombusinessGastric mucosal atrophyReporting systemBiomarkersEuropean Journal of Gastroenterology &amp; Hepatology
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Sex differences in the prevalence of Helicobacter pylori infection: an individual participant data pooled analysis (StoP Project)

2019

Background Helicobacter pylori (H. pylori) infection is more frequent among men, though the magnitude of the association might be inaccurate due to potential misclassification of lifetime infection and publication bias. Moreover, infection is common, and most studies are cross-sectional. Thus, prevalence ratios (PRs) may be easier to interpret than odds ratios (ORs). Aim The aim of this study was to quantify the association between sex and H. pylori infection using controls from 14 studies from the Stomach Cancer Pooling (StoP) Project. Participants and methods H. pylori infection was defined based on IgG serum antibody titers or multiplex serology. Participants were also classified as infe…

Gastritis AtrophicMalemedicine.medical_specialtyconsortiumRisk AssessmentHelicobacter InfectionsSerology03 medical and health sciencesSex Factors0302 clinical medicineAtrophyRisk FactorsInternal medicinePrevalencesexHumansMedicinepooled analysiSerologic TestsStomach cancerAgedHelicobacter pyloriHepatologybiologybusiness.industryStomachGastroenterologyindividual participant dataPublication biasOdds ratioMiddle AgedHelicobacter pyloribiology.organism_classificationmedicine.diseaseAntibodies BacterialConfidence intervalImmunoglobulin G030220 oncology & carcinogenesisMeta-analysisFemale030211 gastroenterology & hepatologyAtrophybusinessEuropean Journal of Gastroenterology &amp; Hepatology
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Addison Disease and Atrophic Gastritis: High Persistent ACTH Levels Although an Adequate Treatment

2014

Settore MED/38 - Pediatria Generale E SpecialisticaAddison Disease Atrophic Gastritis ACTH
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